Health Foods and Multivitamins

Posts Tagged ‘obesity’

We’ve always said that the late dinners go straight to the handles, but no studies to prove it.

This study examined whether the time to go to sleep could be associated with being overweight, as it is known that a few hours sleep is associated with an increased risk of obesity.

52 people participated in the study wore a bracelet to check their daily activities; in addition to pointing in a journal, when and how much they ate and slept for a week. We analyzed two groups: ‘normal sleepers’, where half of the dream of the night was before 5:30 am, and ‘late risers’ when it was later than that time. Based on revised data for 56% of participants were ‘normal sleepers’ and 44% were ‘late risers’. The latter also ate more calories at dinner, and more calories after 8:00 pm, more junk food, more sugary drinks and fewer fruits and vegetables than the ‘normal sleepers’. The ‘late risers’ were to eat 248 calories more per day than normal. This difference was not statistically significant, but the authors say that if the difference persists over time can result in an increase of almost 1 kilo of body weight per month.

That fruit is good we all know, and that gives us a lot of vitamins and minerals needed for proper operation. Within the fruit in this post we will highlight a variety of mandarin. This is because in the press recently published the results of a study which determined that the tangerine was directly related to obesity, since it is a shield against it.

So far, the mandarin was considered the little sister of orange and a snack no more important, but is really much more than that, since in fact a recent study conducted by researchers at the University of Western Ontario has shown that Tangerine contains some substances that will help prevent obesity from childhood. It is therefore important not to overlook this fruit.

Specifically, this study highlights what is the content of the mandarin has nobiletin, a flavonoid found in this fruit and therefore makes it a valuable asset to our health as we help prevent the onset of various diseases. Specifically we will highlight obesity, type two diabetes and atherosclerosis, which is the main cause of various heart diseases.

This study was conducted with rodents, several samples of the same ones who were given a diet high in cholesterol and saturated fats. One group was given as well as food mandarin. This group got much better control levels of blood fat and the effects it had on the body, and especially the mechanism of fat storage is one of the causes of obesity. Even the mice not given mandarin developed fatty liver.

Instead, the sample that was given did not develop fatty liver tangerine or disproportionately accumulated fat. This was due to the flavonoid prevented the accumulation of fat in the liver by a number of substances that trigger the body, improving circulation of fats and avoiding the accumulation of body fat and the formation of a fatty liver, represents a major trigger of obesity.

Is important to take this into account when carrying fruit to the mouth, as tangerine will be a good ally in maintaining the perfect weight and not to go with the fat. At the end of the day the fruit continues to be a strengthening and purifying the body, as evidenced by the mandarin.

that obesity is hereditary (genetic) has been evident even before we knew anything about genes and DNA. Only had to exercise the powers of observation and see how this is concentrated in families.

Thanks to advances in genetic research, we know that one of every 20 cases of morbid obesity, is caused by a specific mutation of a gene and these guys can do little to counteract such genetic determinism. However, for 95% of the morbidly obese and vastly more numerous groups of overweight and obese individuals more ‘normal’, the role of genetics is much more complicated.

In these cases, unlike what happens with monogenic obesity, the genetic component is not deterministic, but only permissive. This means that in these cases, obesity occurs only if they give other triggers such as excess weight are some of the other “deadly sins” that we will be discussing in the coming weeks and are well known ( For example, excessive caloric intake and sedentary lifestyle).

This comes to show that most of us cannot simply shrug our shoulders, sit back (after throwing in the towel) and blame our genes (or our ancestors) or society.

Using a parallel and used by others in similar situations, the genetic mutations linked to common obesity is like a loaded gun. Nothing will happen to it until you pull the trigger. That is, if the individual does not add to the equation of environmental factors leading to obesity, our genes not expressed, as it is evidenced by the much lower obesity rates that existed in previous generations, despite sharing the same genes (see above for Nauru). It is therefore the combination of genetic mutations (present in our genome by tens or hundreds of generations) and an obesogenic environment (appeared in recent decades) that led to the current obesity epidemic.

One might wonder from the evolutionary point of view why we maintain in our genomes many mutations associated with obesity. The normal thing would be that mutations with negative effects were decreased in frequency from one generation to another until the human race disappears.

There are two answers to this question. The first is that most mutations are not expressed until the last decades and, therefore, the selection has not had time to act. The second is that some of these mutations were, paradoxically, vital to the survival of the human species. To understand this we must distance ourselves from the current environment in which we move and think that for thousands of years, and even more recently, food were neither as abundant nor as easy to get (at least in our environment). Therefore, those with mutations that can qualify as ‘saving’, they were able to store and keep more energy (ie fat in adipose tissue) and thus deal with periods of famine to a greater chance of success.

However, in the environment ‘obesogenic’ today, these subjects are at a disadvantage because of their innate predisposition to obesity. This also explains in part why some ethnic groups (eg American Indians or Asians, and especially the inhabitants of the Pacific Islands) have a higher risk of obesity today as cultures that traditionally have been got exposed to famine or food with great physical effort.

So as a summary of this our first ‘cardinal sin’, we say common obesity in the population has a genetic component but are our habits that make the potential locked in our genes is revealed. The bad news is we can not entirely ‘blame’ our genes because we are active accomplices. The good news is that recent research has repeatedly demonstrated that we can eliminate most of the predisposition to obesity with a healthy lifestyle and the ‘penance’ get rid of the consequences of our ‘original sin’.

through a series of articles we will be reviewing the seven ‘deadly sins’ of obesity. The first is undoubtedly the ‘original sin’.

Obesity has become one of the most talked about health issues in newspapers and more avidly investigated in the scientific field. This is not surprising considering that is invading us with such virulence that it is considered an ‘epidemic’, affects 205 million people and 297 million women. Like so many other pandemics in human history, does not respect borders or economic development, which has already received the nickname ‘Globesity. ”

The latest figures published this month in the journal Lancet, show that between 1980 and 2008, the global average body mass index (BMI) has increased each decade by 0.4 kg/m2 in men and 0 5 kg/m2 in women. The most dramatic increase occurs in Oceania, with countries such as Nauru, which already reach the national average BMI of 34.5, well above the threshold of obesity (30.0 kg/m2). During the first half of last century, this area of the world was a different situation, a fact very important to understand why the original sin of obesity.

Rivers of ink have real or virtual discussed at length the reasons for obesity. In fact, the biggest problem we face in the prevention and therapy is the complexity of its etiology. One of the influencing factors will be called as ‘original sin’ which, although it did not originate in time as the Bible itself that precedes each of us as individuals and therefore can do nothing about it. We’re talking of course of the genetic component of obesity that is dependent on our parents and grandparents and the rest of our family tree.

For years the manufacturers of sodas, candy and other processed foods have insisted that corn syrup, high fructose – high fructose corn syrup – is not harmful to health because in the end, sugar is sugar.

But many companies that produce these foods are seeking alternatives to this notorious ingredient, as several studies have linked the corn-based sweetener to the high incidence of diabetes and obesity in the United States.

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